The human organism expresses retroviruses.
They are beneficial particles needed for immune defense, growth, and tissue repair.
Since retroviruses were observed in the first AIDS patients, it has been postulated - and it has since become firmly established dogma - that retroviruses "cause" AIDS. Anti-retroviral drugs are the treatment of choice for those who have tested positive to the protein which is a signature for retroviruses.
Of course killing the retrovirus also deprives us of the repair and defense functions they perform. And the drugs' side effects are often deadly for AIDS patients.
Perhaps to re-think our approach in this would be a good idea?
Do White Blood Cells use Retroviruses for Cell fusions?
by Cal Crilly
Alveolar macrophages (white blood cells) produce the Env protein of a human endogenous retrovirus, HERV-E 4-1, in a subgroup of interstitial lung diseases.
Ok I'll try and explain where the research is going and why retroviruses are also likely to appear in white blood cells or macrophages in response to real infections of bacteria and fungi (the retroviruses being functional and already in the macrophage/WBC DNA.)
The white blood cell monocytes or baby white blood cells join together into what they call 'giant cell multinucleated cells', this is in response to large infections, lots of dead cell debris, foreign bodies lodged in the body, anything that needs to be removed etc.
So baby monocyte cells look like they join together into what I call "Giant death star white blood cells" or macrophages made of many immature white blood cell monocytes bound together with retroviruses creating cell fusions.
TB and malaria are prime cases where giant cells are seen as a response to remove quite big bugs.
"Macrophages undergo fusion with other macrophages to form the hallmark multinucleated giant cells of chronic inflammation. However, neither the existence of distinct morphological types of giant cells, the signaling pathways that induce their formation, the molecular mechanism(s) of macrophage fusion, nor the significance of macrophage multinucleation at chronic inflammatory sites are well understood. Our efforts have been focused on these unknowns, particularly as they relate to the foreign body-type giant cells that form on implanted biomaterials and biomedical devices"
Macrophage fusion and multinucleated giant cells of inflammation.