I had heard of using hydrogen peroxide before, especially as an intravenous infusion. Ed McCabe, who wrote Flood Your Body with Oxygen was thrown in prison for advocating its use. Big Pharma's shock troops, the quackbusters, have set their sights on yet another doctor, South Carolina physician James Shortt, M.D., using peroxide with his patients, as reported by Chris Gupta in this article.
Educate-Yourself has a good summary on using hydrogen peroxide and what it does for your health, but just today I read an account that I would like to share, as it's a real personal experience, the kind of story we can all relate to, and perhaps learn from.
Bill Munro says that using hydrogen peroxide in the ways normally suggested did not appeal to him. He was not keen to take baths in it, drink it or get the intravenous infusions. So he decided to inhale 3% peroxide, using a nasal spray pump. He also used peroxide in his garden and much to his delight, the bugs vanished while his veggies grew larger and healthier than before.
"Hydrogen Peroxide is the most over looked chemical used by man. New uses of it are coming to light every day. Bad bugs be they, in your garden or your body, they cannot live in an oxygen rich environment."
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Additional information received by email through Barbara Boss, quoting Bill Munro:
The first thing I had to know about Hydrogen Peroxide was, how much and when to inhale. It has taken 13 years to set up a schedule. Many times I would have a uncomfortable throat from too much peroxide. I would have several people tell me the same thing. I have now worked out for the past three weeks. I inhale through my mouth into my lungs the 3% peroxide that I bought at the drug store. I inhale 7 times a day. Breakfast, between, lunch, between, dinner, between, and at bedtime. And if I get up in the night I inhale then. With each inhale I pump 5 times. All the discomforts are gone. All of my functions seem to be normal. I will be going to the doctor in a couple days, for some kind of checks. I will keep you posted.
The article below has confirmed what I said for 13 years. Think positive.
Studies Reveal How Cells Sense Oxygen
Three studies in the June issue of Cell Metabolism offer additional insight into how the cells of mammals sense oxygen. Oxygen plays a central role in fueling cells, and oxygen deficiency underlies many disease conditions, including heart attack, stroke, inflammation, and cancer.
The findings should help to resolve a long-standing controversy over the identity of mammalian oxygen sensors, according to the researchers.
The studies report genetic evidence that the cellular powerhouses known as mitochondria are required for cells to detect and respond to changes in the availability of oxygen. Byproducts of mitochondrial activity, called reactive oxygen species or free radicals, signal low oxygen conditions, launching a cascade of effects that allow cells to adapt, their research shows.
"Oxygen is so central for life," said M. Celeste Simon of the University of Pennsylvania, an author on two of the three studies. "Oxygen deprivation is behind stroke, heart attack, inflammation, and tumor physiology. Even brief oxygen limitation can render someone unable to function.
"While cellular responses to low oxygen have been studied extensively, the precise identity of mammalian oxygen sensors remains controversial. The current studies address the controversy about the role that mitochondria play in oxygen sensing, which should allow the field to move forward."
Mammals require oxygen to obtain energy and respond to decreases in oxygen supply by activating genes that preserve oxygen in tissues. Earlier findings suggested that reactive oxygen species generated by mitochondria under low oxygen conditions regulate a variety of hypoxic responses, including the activation of hypoxia-inducible factor 1 (HIF-1). HIF-1 is a master regulator of local cellular and systemic responses to oxygen starvation.
In the new studies, the groups used a variety of genetic methods to specifically disable the function of mitochondrial components, thereby inhibiting mitochondrial activity and the production of its free radical byproducts, including hydrogen peroxide. They then examined the consequences of that inactivation for the cell's defenses against low oxygen.
In mouse cells lacking mitochondrial activity, oxygen sensing fails and a component of element of hypoxia response, HIF-1a, is degraded as it is under normal oxygen, Simon's team reported. Treatment with hydrogen peroxide stabilized the defense protein.
Two companion studies in the same journal issue, led by Paul Schumacker at The University of Chicago and Navdeep Chandel at Northwestern University, provide additional support that reactive oxygen species produced by mitochondria under conditions of low oxygen stabilize HIF-1a.
The new findings bolster an earlier report by the studies' senior authors, which implicated mitochondria as potential oxygen sensors. That report had remained controversial because experiments designed to test the idea by limiting mitochondrial function with nonspecific drugs yielded conflicting results.
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